Galantamine protects against beta amyloid peptide-induced DNA damage in a model for Alzheimer's disease
Pages: 916-917
Year: 2017 Issue: 6
Journal: Neural Regeneration Research
Abstract: Alzheimer's disease (AD) is the most common type of dementia in elderly population. With a growing aging population not only in the United States but also in the worldwide, AD constitutes an emergent public health problem. Over decades, the prevailing hypothesis was that neurodegeneration might result from one or two of the specific lesions that characterize AD, e.g., accumulation of amyloid plaques (extracellular deposits of amyloid-beta peptide (Aβ1–42) and hyperphosphorylation of tau protein.
Year: 2017 Issue: 6
Journal: Neural Regeneration Research
Abstract: Alzheimer's disease (AD) is the most common type of dementia in elderly population. With a growing aging population not only in the United States but also in the worldwide, AD constitutes an emergent public health problem. Over decades, the prevailing hypothesis was that neurodegeneration might result from one or two of the specific lesions that characterize AD, e.g., accumulation of amyloid plaques (extracellular deposits of amyloid-beta peptide (Aβ1–42) and hyperphosphorylation of tau protein.
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