Similar articles for PMID: 25826266

Year	Number of Results
1970	1
1999	1
2002	1
2003	1
2005	2
2007	4
2008	8
2009	4
2010	3
2011	4
2012	5
2013	12
2014	5
2015	7
2016	14
2017	9
2018	12
2019	10
2020	14
2021	15
2022	18
2023	3
2024	0

1

Network analyses reveal novel aspects of ALS pathogenesis.

Sanhueza M, Chai A, Smith C, McCray BA, Simpson TI, Taylor JP, Pennetta G. Sanhueza M, et al. PLoS Genet. 2015 Mar 31;11(3):e1005107. doi: 10.1371/journal.pgen.1005107. eCollection 2015 Mar. PLoS Genet. 2015. PMID: 25826266 Free PMC article.

2

hVAPB, the causative gene of a heterogeneous group of motor neuron diseases in humans, is functionally interchangeable with its Drosophila homologue DVAP-33A at the neuromuscular junction.

Chai A, Withers J, Koh YH, Parry K, Bao H, Zhang B, Budnik V, Pennetta G. Chai A, et al. Hum Mol Genet. 2008 Jan 15;17(2):266-80. doi: 10.1093/hmg/ddm303. Epub 2007 Oct 18. Hum Mol Genet. 2008. PMID: 17947296 Free PMC article.

3

Accumulation of wildtype and ALS-linked mutated VAPB impairs activity of the proteasome.

Moumen A, Virard I, Raoul C. Moumen A, et al. PLoS One. 2011;6(10):e26066. doi: 10.1371/journal.pone.0026066. Epub 2011 Oct 5. PLoS One. 2011. PMID: 21998752 Free PMC article.

4

Insights into ALS pathomechanisms: from flies to humans.

Chai A, Pennetta G. Chai A, et al. Fly (Austin). 2015;9(2):91-8. doi: 10.1080/19336934.2015.1114694. Fly (Austin). 2015. PMID: 26594942 Free PMC article.

5

Motor neuron apoptosis and neuromuscular junction perturbation are prominent features in a Drosophila model of Fus-mediated ALS.

Xia R, Liu Y, Yang L, Gal J, Zhu H, Jia J. Xia R, et al. Mol Neurodegener. 2012 Mar 24;7:10. doi: 10.1186/1750-1326-7-10. Mol Neurodegener. 2012. PMID: 22443542 Free PMC article.

6

Increased levels of phosphoinositides cause neurodegeneration in a Drosophila model of amyotrophic lateral sclerosis.

Forrest S, Chai A, Sanhueza M, Marescotti M, Parry K, Georgiev A, Sahota V, Mendez-Castro R, Pennetta G. Forrest S, et al. Hum Mol Genet. 2013 Jul 1;22(13):2689-704. doi: 10.1093/hmg/ddt118. Epub 2013 Mar 13. Hum Mol Genet. 2013. PMID: 23492670 Free PMC article.

7

A Drosophila model of ALS: human ALS-associated mutation in VAP33A suggests a dominant negative mechanism.

Ratnaparkhi A, Lawless GM, Schweizer FE, Golshani P, Jackson GR. Ratnaparkhi A, et al. PLoS One. 2008 Jun 4;3(6):e2334. doi: 10.1371/journal.pone.0002334. PLoS One. 2008. PMID: 18523548 Free PMC article.

8

Different gene expression profiles in iPSC-derived motor neurons from ALS8 patients with variable clinical courses suggest mitigating pathways for neurodegeneration.

Oliveira D, Morales-Vicente DA, Amaral MS, Luz L, Sertié AL, Leite FS, Navarro C, Kaid C, Esposito J, Goulart E, Caires L, Alves LM, Melo US, Figueiredo T, Mitne-Neto M, Okamoto OK, Verjovski-Almeida S, Zatz M. Oliveira D, et al. Hum Mol Genet. 2020 Jun 3;29(9):1465-1475. doi: 10.1093/hmg/ddaa069. Hum Mol Genet. 2020. PMID: 32280986

9

Imbalance of mitochondrial dynamics in Drosophila models of amyotrophic lateral sclerosis.

Altanbyek V, Cha SJ, Kang GU, Im DS, Lee S, Kim HJ, Kim K. Altanbyek V, et al. Biochem Biophys Res Commun. 2016 Dec 9;481(3-4):259-264. doi: 10.1016/j.bbrc.2016.10.134. Epub 2016 Oct 31. Biochem Biophys Res Commun. 2016. PMID: 27810362

10

SOD1 activity threshold and TOR signalling modulate VAP(P58S) aggregation via reactive oxygen species-induced proteasomal degradation in a Drosophila model of amyotrophic lateral sclerosis.

Chaplot K, Pimpale L, Ramalingam B, Deivasigamani S, Kamat SS, Ratnaparkhi GS. Chaplot K, et al. Dis Model Mech. 2019 Feb 7;12(2):dmm033803. doi: 10.1242/dmm.033803. Dis Model Mech. 2019. PMID: 30635270 Free PMC article.

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